Knowledge about the BG functions may have important implications for the area of linguistics in general and SLA in particular. This may be why BG-lesion patients cannot produce their first language in spite of their intact declarative memory system. In contrast, a first language may have been almost completely proceduralized without leaving much of a trace in the declarative system. When a patient suffers a BG lesion, the parts of the second language that have already been proceduralized wil be damaged, but other parts of SL that have not been proceduralized will be preserved. Although the automatization of a second language through the BD is ongoing, it may not be complete. The interesting fact that the patients’ second languages are better preserved may imply that their second languages are processed more by the declarative memory system. Additionally a polyglot’s more fluent language tends to be more seriously damaged than a less fluent language. According to Fabbro (1999), basal ganglia aphasics develop symptoms such as reduced voice volume, foreign accent syndrome, perseveration (involuntary repetition of words, syllables etc.), and agrammatism. “Aphasic syndromes caused by BG (basal ganglia) lesions indicate what roles the BG may play in language functions. Excerpts from “The Neurobiology of Learning”īy John H.
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